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Human Molecular Genetics Advance Access originally published online on September 14, 2007
Human Molecular Genetics 2007 16(24):3071-3080; doi:10.1093/hmg/ddm265
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© The Author 2007. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Heterozygosity of mannose-binding lectin (MBL2) genotypes predicts advantage (heterosis) in relation to fatal outcome in intensive care patients

Dorthe Hellemann1,2, Anders Larsson6, Hans O. Madsen2, Jan Bonde3, Jens Otto Jarløv4, Jørgen Wiis3, Torsten Faber1, Jørn Wetterslev5 and Peter Garred2,*

1 Department of Anaesthesiology and Intensive Care, Herlev,, 2 Department of Clinical Immunology, sect.7631,, 3 Intensive Care Unit 4131,, 4 Department of Clinical Microbiology, Herlev,, 5 Copenhagen Trial Unit, Copenhagen University Hospital, Rigshospitalet, Denmark and 6 Clinical Institute, Århus University, Denmark

* To whom correspondence should be addressed at: Department of Clinical Immunology, sect. 7631, Copenhagen University Hospital, Rigshospitalet, Blegdamsvej 9, 2100 Copenhagen O, Denmark. Tel: +45 35457631; Fax: +45 35398766; Email: garred{at}post5.tele.dk

Received June 22, 2007; Accepted August 25, 2007

Polymorphisms in the MBL2 gene, which affect the structure and influence on the serum concentration of mannose-binding lectin (MBL), are associated with inflammatory and infectious conditions. The importance of MBL2 polymorphisms on outcome in critical ill patients is unclear. Five hundred and thirty-two consecutive critically ill patients admitted to an intensive care unit (ICU) were included over a period of 18 months. Five hundred and thirty-three individuals served as controls. Vital status was obtained 15.5 months after the last patient was included. MBL2 polymorphisms were determined by a PCR-based assay. Homozygosity for MBL2 variant alleles (O/O) causing MBL structural defects was associated with the highest adjusted mortality rate followed by homozygosity for the normal MBL2 allele (A/A) encoding high MBL levels, whereas heterozygous A/O patients had the most favourable outcome (P = 0.015). MBL2 alleles were not associated with death in ICU (n = 166, P = 0.7), but the association appeared soon after discharge from ICU (n = 366): hazard ratio (HR) for O/O using A/A as reference was 1.33 (95% CI: 0.8–2.2) and for A/O it was 0.62 (95% CI: 0.4–0.8) respectively (P = 0.0045) at completion. No difference in MBL2 frequency was observed between patients and controls at baseline, and between patients classified as having sepsis or not. However, patients with the MBL2 O/O genotype had an increased frequency of Gram-positive bacterial infection (P = 0.01). Heterozygosity for MBL2 alleles confers a protective effect whereas homozygosity is associated with the worst outcome soon after discharge from ICU. This may be an example of heterosis.


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