Corrigendum
for Pichierri et al., Hum. Mol. Genet. 11 (21) 2531-2546.
Human Molecular Genetics, 2004, Vol. 13, No. 12 1289
DOI: 10.1093/hmg/ddh148
Human Molecular Genetics, Vol. 13, No. 12 © Oxford University Press 2004; all rights reserved
DNA cross-link-dependent RAD50/MRE11/NBS1 subnuclear assembly requires the Fanconi anemia C protein
Pietro Pichierri,
Dietrich Averbeck and
Filippo Rosselli
Human Molecular Genetics 11, 2531–2546 (2002)
The authors apologise for including in the text the wrong figure representing the phosphorylation of NBS1 after ICL in FA cells. This figure was picked up by mistake from a blot representing NBS1 phosphorylation in BS cells and already published in the J. Cell Biol., 2002 157, 19–30. The two manuscripts were edited at the same time before their initial submission and the two blots were mixed up and confused because of their similarity. We now include here the figure presenting the correct data on the NBS1 phosphorylation in FA-C cells following ICL induction.
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Figure 3. Western blot analysis of NBS1 and MRE11 proteins. (A) Mobility shift of NBS1 in response to DNA damage in normal, FA-C and FA-C corrected cells. In treated samples, proteins were isolated from cells 3 h after exposure to ionizing radiation (5 Gy) or MMC (0.1 µg/ml/h). NBS1 is equally expressed in all cell lines examined independently of the FA status or of the treatment, but it shows altered NBS1 migration in MMC-exposed FA-C cells compared with the other two cell lines. Ponceau Red staining of the membrane monitored the equal loading of protein in each slot.
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